The alternate explanation is that arteries are damaged by the entry of only one form of lipoprotein, the small, dense LDL-cholesterol particles, and then only if those small, dense LDL-cholesterol particles have been damaged by becoming oxidized. It is proposed that oxidized small, dense LDL-cholesterol particles have the ability to enter damaged arteries where they become ‘stuck’ within the arterial wall, inducing an inflammatory reaction that leads ultimately to the irreversible arterial damage recognized as the arterial plaque. Thus according to the explanation it is not ‘cholesterol’ which
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